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Cardiac KV Channel Antibody Explorer Kit

A Screening Package of Cardiac KV Channel Antibodies Economically Priced
  • Lyophilized Powder
  • Antigen Incl.
Cat #: AK-320
Sizes: 36 Vials
Last update: 24/01/2020

Alomone Labs is pleased to offer the Cardiac KV Channel Antibody Explorer Kit (#AK-320). This Explorer Kit includes cardiac KV channel antibodies with their respective peptide control antigen. An ideal tool for screening purposes.

For research purposes only, not for human use
Compounds
Product NameCat #Size
Anti-KV1.4 Antibody
 APC-007 1 x 50 µl
Anti-KV1.5 (KCNA5) Antibody
 APC-004 1 x 50 µl
Anti-KV1.5 (KCNA5) (extracellular) Antibody
 APC-150 1 x 50 µl
Anti-Mouse KV1.7 (KCNA7) Antibody
 APC-063 1 x 50 µl
Anti-KV3.1b (KCNC1) Antibody
 APC-014 1 x 50 µl
Anti-KCNC4 (KV3.4) Antibody
 APC-019 1 x 50 µl
Anti-KV4.2 Antibody
 APC-023 1 x 50 µl
Guinea pig Anti-KV4.2 Antibody
 AGP-038 1 x 50 µl
Anti-KV4.3 Antibody
 APC-017 1 x 50 µl
Anti-KChIP2 Antibody
 APC-142 1 x 50 µl
Anti-KCNE1 (IsK) Antibody
 APC-163 1 x 50 µl
Anti-KCNE2 (MiRP1) Antibody
 APC-054 1 x 50 µl
Anti-KCNE3 (MiRP2) Antibody
 APC-118 1 x 50 µl
Anti-KCNQ1 Antibody
 APC-022 1 x 50 µl
Anti-KCNQ1 (extracellular) Antibody
 APC-168 1 x 50 µl
Anti-KCNH2 (erg1) Antibody
 APC-016 1 x 50 µl
Anti-KCNH2 (HERG) Antibody
 APC-062 1 x 50 µl
Anti-KCNH2 (HERG) (extracellular) Antibody
 APC-109 1 x 50 µl
Note A guinea pig polyclonal antibody (#AGP-038) is included in this Explorer Kit. Please take into account when reacting with a secondary antibody.
Scientific Background

    • Voltage-gated K+ channels (KV) have a significant role in numerous cellular processes including regulation of apoptosis, cell growth and differentiation, and maintenance of cardiac activity. 

      Cardiac KV channels are also known as hERG channels and are involved in action potential (AP) propagation in cardiac myocytes. The AP is elicited by inward sodium current and rapid depolarization of the membrane. Repolarization is divided into several phases - an initial rapid and short depolarization is followed by a much slower “plateau” phase that ensures sufficient time for entry of extracellular Ca2+ into the myocyte for optimal excitation-contraction coupling. In addition, delayed repolarization also makes cardiac muscle refractory to premature excitation - an important safeguard against the generation of re-entrant arrhythmia. This phase is prolonged because the potassium influx through the hERG channel is slow to activate and is reduced at positive membrane potentials1,2.

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